Relief Of Mild Joint Pain And Inflammation.
Ingredients in Inflavonoid Intensive Care have been traditionally used or shown:
- For temporary relief of pain and joint inflammation.1
- Contains BCM-95TM Turmeric.2
- A condition specific BCM-95TM product for use in acute and painful conditions.
Directions
Directions for use (Adults): For symptomatic relief of mild joint pain and mild arthritis:Take 1 tablet every 2 hours (6 tablets per day).
To relieve inflammation:Take 2 tablets twice daily.
Ingredients
|
Each tablet contains: |
|
|
Quercetin |
250 mg |
| Citrus bioflavonoids extract | 100 mg |
|
Capsicum frutescens, fruit powder |
25 mg |
| Dry herbal extracts: | |
| Curcuma longa (BCM-95™ Turmeric), rhizome | 152 mg |
| Derived from dry rhizome | 3.8 g |
| Equivalent Curcumin | 106 mg |
| Boswellia serrata (BosPure® Boswellia), gum oleoresin |
150 mg |
| Derived from dry gum oleoresin | 1.5 g |
| Salix alba (White willow), stem bark | 60 mg |
| Derived from dry stem bark | 1.5 g |
| Zingiber officinale (Ginger), rhizome | 62.5 mg |
| Derived from dry rhizome | 500 mg |
Free from animal products, dairy protein, lactose, eggs, gluten, wheat, nuts, yeast, soy protein and salt. Free from artificial colours, sweeteners, flavours and preservatives.
Relieves acute inflammation: The ingredients in Inflavonoid Intensive Care work at various points within the inflammatory cascade to decrease inflammation and pain associated with mild arthritis via the following mechanisms:
- Boswellia inhibits 5-lipoxygenase (5-LOX).1
- White willow bark inhibits prostaglandin synthesis by inhibiting cyclooxygenase (COX-2) mediated prostaglandin release shown in vitro.
- Ginger suppresses prostaglandin synthesis through inhibition of COX and LOX.
- Quercetin inhibits the activation of NF-ƙB demonstrated in vitro studies.
Volatile oils, naturally found in BCM-95 turmeric, contribute to its effect. These oils exhibit anti-inflammatory activity with demonstrated ability to bind to the nuclear receptor, perisome proliferator-activated receptor γ (PPARγ), inhibit prostaglandin E2 (PGE2) production and inhibit inducible nitric oxide production during the inflammation process.
